The mitochondrial-derived peptide MOTS-c suppresses ferroptosis and alleviates acute lung injury induced by myocardial ischemia reperfusion via PPARγ signaling pathway-Reference-Cited by-同舟云学术

The mitochondrial-derived peptide MOTS-c suppresses ferroptosis and alleviates acute lung injury induced by myocardial ischemia reperfusion via PPARγ signaling pathway

Published:2023-08 Issue: Volume:953 Page:175835
ISSN:0014-2999
Container-title:European Journal of Pharmacology
language:en
Short-container-title:European Journal of Pharmacology

Author:

Lu Peng,Li Xiaopei,Li Ben,Li Xiangyu,Wang Chufan,Liu Zhaoyang,Ji Yumeng,Wang Xufeng,Wen Ziang,Fan Jidan,Yi Chenlong,Song Meijuan,Wang Xiaowei

Publisher

Elsevier BV

Subject

Pharmacology

Reference51 articles.

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1. Effects of empagliflozin and dapagliflozin on serum humanin, MOTS-c levels, nitrosative stress, and ferroptosis parameters in diabetic patients with heart failure;European Journal of Pharmacology;2024-11

2. Relationship between ferroptosis and mitophagy in acute lung injury: a mini-review;PeerJ;2024-09-10

3. The role of FPR2-mediated ferroptosis in formyl peptide-induced acute lung injury against endothelial barrier damage and protective effect of the mitochondria-derived peptide MOTS-c;International Immunopharmacology;2024-04

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