Opposing effects of cyclooxygenase-2 selective inhibitors on oxygen-glucose deprivation-induced neurotoxicity-Reference-Cited by-同舟云学术

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Opposing effects of cyclooxygenase-2 selective inhibitors on oxygen-glucose deprivation-induced neurotoxicity

Published:2004-06 Issue:1-3 Volume:493 Page:45-55
ISSN:0014-2999
Container-title:European Journal of Pharmacology
language:en
Short-container-title:European Journal of Pharmacology

Author:

Gendron Tania F.,Brunette Eric,Mealing Geoffrey A.R.,Nguyen Adele,Tauskela Joseph S.,Morley Paul

Funder

Natural Sciences and Engineering Research Council of Canada

National Research Council Canada

Heart and Stroke Foundation of Canada

Canadian Stroke Network

Publisher

Elsevier BV

Subject

Pharmacology

Reference39 articles.

1. Cyclooxygenase-2 selective inhibitors aggravate kainic acid induced seizure and neuronal cell death in the hippocampus;Baik;Brain Res.,1999

2. Protaglandins stimulate Ca2+-dependent glutamate release in astrocytes;Bezzi;Nature,1998

3. N-methyl-d-aspartate- or glutamate-mediated toxicity in cultured rat cortical neurons is antagonized by FPL 15896AR;Black;J. Neurochem.,1995

4. NS-398: cyclooxygenase-2 independent inhibition of leukocyte priming for lipid body formation and enhanced leukotriene generation;Bozza;Prostaglandins Leukot. Essent. Fat. Acids,2002

5. Long-term elevation of cyclooxygenase-2, but not lipoxygenase, in regions synaptically distant from spreading depression;Caggiano;J. Comp. Neurol.,1996

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2. Synaptic and Extrasynaptic NMDA Receptors Differentially Modulate Neuronal Cyclooxygenase-2 Function, Lipid Peroxidation, and Neuroprotection;Journal of Neuroscience;2011-09-28

3. Oxygen Glucose Deprivation Model of Cerebral Stroke in PC-12 Cells: Glucose as a Limiting Factor;Toxicology Mechanisms and Methods;2009-02

4. Selective increase of neuronal cyclooxygenase-2 (COX-2) expression in vulnerable brain regions of rats with experimental Wernicke’s encephalopathy: effect of nimesulide;Metabolic Brain Disease;2008-05-15

5. Cyclooxygenase Inhibition in Ischemic Brain Injury;Current Pharmaceutical Design;2008-05-01

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