Two distinct dysfunctions in diabetic mouse mesenteric artery contraction are caused by changes in the Rho A–Rho kinase signaling pathway-Reference-Cited by-同舟云学术

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Two distinct dysfunctions in diabetic mouse mesenteric artery contraction are caused by changes in the Rho A–Rho kinase signaling pathway

Published:2012-05 Issue:1-3 Volume:683 Page:217-225
ISSN:0014-2999
Container-title:European Journal of Pharmacology
language:en
Short-container-title:European Journal of Pharmacology

Author:

Nobe Koji,Hashimoto Terumasa,Honda Kazuo

Funder

Grant-in-Aid for the Encouragement of Young Scientists from MEXT

“High-Tech Research Center” Project for Private Universities with a matching fund subsidy from MEXT

Publisher

Elsevier BV

Subject

Pharmacology

Reference25 articles.

1. Direct vasodilative effect of FK506 on porcine mesenteric artery in small bowel transplantation;Akiyoshi;J. Pediatr. Surg.,2009

2. High glucose concentration augments angiotensin II mediated contraction via AT1 receptors in rat thoracic aorta;Arun;Pharmacol. Res.,2004

3. Comparison of skin microvascular reactivity with hemostatic markers of endothelial dysfunction and damage in type 2 diabetes;Beer;Vasc. Health Risk Manag.,2008

4. Elevated epidermal growth factor receptor phosphorylation induces resistance artery dysfunction in diabetic db/db mice;Belmadani;Diabetes,2008

5. Cardiovascular complications of diabetes mellitus: the tissue factor perspective;Bogdanov;Thromb. Res.,2010

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3. Hirudin prevents vascular endothelial cell apoptosis and permeability enhancement induced by the serum from rat with chronic renal failure through inhibiting RhoA / ROCK signaling pathway;Drug Development Research;2020-12-20

4. Abnormal Ca2+ handling contributes to the impairment of aortic smooth muscle contractility in Zucker diabetic fatty rats;Journal of Molecular and Cellular Cardiology;2020-04

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