Chronic ethanol consumption increases vascular oxidative stress and the mortality induced by sub-lethal sepsis: Potential role of iNOS

Author:

Ceron Carla S.,do Vale Gabriel T.,Simplicio Janaina A.,Ricci Sthefany T.,De Martinis Bruno S.,de Freitas Andressa,Tirapelli Carlos R.

Funder

Conselho Nacional de Desenvolvimento Científico e Tecnológico, Brazil

Fundação de Amparo à Pesquisa do Estado de São Paulo, Brazil

Publisher

Elsevier BV

Subject

Pharmacology

Reference43 articles.

1. Effects of chronic ethanol consumption in experimental sepsis;Barros;Alcohol Alcohol.,2012

2. Inhibition of leukocyte rolling by nitric oxide during sepsis leads to reduced migration of active microbicidal neutrophils;Benjamim;Infect. Immun.,2002

3. Role of nitric oxide in the failure of neutrophil migration in sepsis;Benjamim;J. Infect. Dis.,2000

4. Vasoplegia in sepsis depends on the vascular system, vasopressor, and time-point: a comparative evaluation in vessels from rats subjected to the cecal ligation puncture model;Bernardelli;Can. J. Physiol. Pharmacol.,2016

5. Impaired vascular function in sepsis-surviving rats mediated by oxidative stress and Rho-Kinase pathway;De Souza;Redox Biol.,2016

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