Inhibition of the nitric oxide/cyclic guanosine monophosphate pathway limited the cardioprotective effect of post-conditioning in hearts with apical myocardial infarction

Author:

Correa Francisco,Buelna-Chontal Mabel,Chagoya Victoria,García-Rivas Gerardo,Vigueras Rosa María,Pedraza-Chaverri José,García-Niño Wylly Ramsés,Hernández-Pando Rogelio,León-Contreras Juan Carlos,Zazueta Cecilia

Funder

National Council of Science and Technology

Publisher

Elsevier BV

Subject

Pharmacology

Reference42 articles.

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2. Sequential changes of energy metabolism and mitochondrial function in myocardial infarction induced by isoproterenol in rats: a long-term and integrative study;Chagoya de Sánchez;Can. J. Physiol. Pharmacol.,1997

3. Accelerated phospholipid degradation and associated membrane dysfunction in irreversible, ischemic liver cell injury;Chien;J. Biol. Chem.,1978

4. Relationship between oxidative stress and mitochondrial function in the post-conditioned heart;Correa;J. Bioenerg. Biomembr.,2008

5. Mitochondrial permeability transition relevance for apoptotic triggering in the post-ischemic heart;Correa;Int. J. Biochem. Cell Biol.,2007

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