Vorinostat upregulates MICA via the PI3K/Akt pathway to enhance the ability of natural killer cells to kill tumor cells

Author:

Xia Chenglai,He Zhihong,Cai Yantao,Liang Shaofen

Funder

Medical Project of Science and Technology Department of Foshan

Science and Technology Bureau of Foshan

Foshan anti-tumor innovative drug research and engineering technology center

Foshan Medicine Dengfeng Project of China

Publisher

Elsevier BV

Subject

Pharmacology

Reference46 articles.

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3. Vorinostat, a pan-HDAC inhibitor, abrogates productive HPV-18 DNA amplification;Banerjee;Proc. Natl. Acad. Sci. U.S.A.,2018

4. Activation of NK cells and T cells by NKG2D, a receptor for stress-inducible MICA;Bauer;Science,1999

5. Methodological insights for randomized clinical trials of retinitis pigmentosa: lessons learned from a trial of ValproicAcid;Brooks;JAMAOphthalmol,2018

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