Modulation of KCNQ1 alternative splicing regulates cardiac IKs and action potential repolarization
Author:
Publisher
Elsevier BV
Subject
Physiology (medical),Cardiology and Cardiovascular Medicine
Reference31 articles.
1. Rate dependency of delayed rectifier currents during the guinea-pig ventricular action potential;Rocchetti;J Physiol,2001
2. Accumulation of slowly activating delayed rectifier potassium current (IKs) in canine ventricular myocytes;Stengl;J Physiol,2003
3. Subunit interaction determines IKs participation in cardiac repolarization and repolarization reserve;Silva;Circulation,2005
4. Probing the contribution of IKs to canine ventricular repolarization: key role for β-adrenergic receptor stimulation;Volders;Circulation,2003
5. Prominent IKs in epicardium and endocardium contributes to development of transmural dispersion of repolarization but protects against development of early afterdepolarizations;Burashnikov;J Cardiovasc Electrophysiol,2002
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1. Neurohormonal Regulation of I Ks in Heart Failure: Implications for Ventricular Arrhythmogenesis and Sudden Cardiac Death;Journal of the American Heart Association;2020-09-15
2. Long Noncoding RNA Kcna2 Antisense RNA Contributes to Ventricular Arrhythmias via Silencing Kcna2 in Rats With Congestive Heart Failure;Journal of the American Heart Association;2017-12-02
3. Pro-arrhythmogenic Effects of the V141M KCNQ1 Mutation in Short QT Syndrome and Its Potential Therapeutic Targets: Insights from Modeling;Journal of Medical and Biological Engineering;2017-07-05
4. Transmural gradients in ion channel and auxiliary subunit expression;Progress in Biophysics and Molecular Biology;2016-12
5. Modulation of potassium channel KCNQ1 transcript in right atrial appendage of patients with postoperative atrial fibrillation;International Journal of Cardiology;2016-11
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