U-waves and T-wave peak to T-wave end intervals in patients with catecholaminergic polymorphic ventricular tachycardia, effects of beta-blockers
Author:
Publisher
Elsevier BV
Subject
Physiology (medical),Cardiology and Cardiovascular Medicine
Reference22 articles.
1. Mutations in the cardiac ryanodine receptor gene (hRyR2) underlie catecholaminergic polymorphic ventricular tachycardia;Priori;Circulation,2001
2. Arrhythmic disorder mapped to chromosome 1q42-q43 causes malignant polymorphic ventricular tachycardia in structurally normal hearts;Swan;J Am Coll Cardiol,1999
3. Catecholaminergic polymorphic ventricular tachycardia: recent mechanistic insights;Kontula;Cardiovasc Res,2005
4. Molecular and electrophysiological bases of catecholaminergic polymorphic ventricular tachycardia;Uwais;J Cardiovasc Electrophysiol,2007
5. Cellular mechanism of calcium-mediated triggered activity in the heart;Katra;Circ Res,2005
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2. Genealogy and clinical course of catecholaminergic polymorphic ventricular tachycardia caused by the ryanodine receptor type 2 P2328S mutation;PLOS ONE;2020-12-14
3. Importance of electrocardiographic markers in predicting cardiac events in children;Biomarkers in Medicine;2020-12
4. Identification of a novel exon3 deletion of RYR2 in a family with catecholaminergic polymorphic ventricular tachycardia;Annals of Noninvasive Electrocardiology;2019-01-07
5. Prolonged T-wave peak-end interval in Down syndrome patients with congenitally normal hearts;Pediatrics International;2018-06
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