Fanconi anemia and Bloom's syndrome crosstalk through FANCJ–BLM helicase interaction
Author:
Publisher
Elsevier BV
Subject
Genetics
Reference70 articles.
1. Expanded roles of the Fanconi anemia pathway in preserving genomic stability;Kee;Genes Dev.,2010
2. Molecular genetics of RecQ helicase disorders;Hanada;Cell Mol. Life Sci.,2007
3. The Bloom's syndrome gene product is homologous to RecQ helicases;Ellis;Cell,1995
4. BLM has early and late functions in homologous recombination repair in mouse embryonic stem cells;Chu;Oncogene,2010
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1. Response to Replication Stress and Maintenance of Genome Stability by WRN, the Werner Syndrome Protein;International Journal of Molecular Sciences;2024-07-30
2. The DNA helicase FANCJ (BRIP1) functions in double strand break repair processing, but not crossover formation during prophase I of meiosis in male mice;PLOS Genetics;2024-02-20
3. The DNA helicase FANCJ (BRIP1) functions in Double Strand Break repair processing, but not crossover formation during Prophase I of meiosis in male mice;2023-10-08
4. The BLM helicase is a new therapeutic target in multiple myeloma involved in replication stress survival and drug resistance;Frontiers in Immunology;2022-12-09
5. G-Quadruplex-Binding Proteins: Promising Targets for Drug Design;Biomolecules;2022-04-29
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