Urinary Diversion Results in Marked Decreases in Proliferation and Apoptosis in Fetal Bladder
Author:
Affiliation:
1. Department of Anatomy and Cell Biology, University of Pennsylvania, Philadelphia, Pennsylvania, and Division of Urology, Albany Medical College, Albany, New York (BK)
Publisher
Ovid Technologies (Wolters Kluwer Health)
Subject
Urology
Reference26 articles.
1. The Developing Human: Clinically Oriented Embryology;Moore,1998
2. Measurement of fetal urine production in normal pregnancy by real-time ultrasonography;Rabinowitz;Am J Obstet Gynecol,1989
3. Mechanical stretch regulates cell survival in human bladder smooth muscle cells in vitro;Galvin;Am J Physiol Renal Physiol,2002
4. Smooth muscle differentiation and cell turnover in mouse detrusor development;Smeulders;J Urol,2002
5. Urinary outflow obstruction increases apoptosis and deregulates Bcl-2 and Bax expression in the fetal ovine bladder;Thiruchelvam;Am J Pathol,2003
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1. Simulated physiological stretch increases expression of extracellular matrix proteins in human bladder smooth muscle cells via integrin α4/αv-FAK-ERK1/2 signaling pathway;World Journal of Urology;2016-12-24
2. Future Perspectives in Bladder Tissue Engineering;Current Bladder Dysfunction Reports;2015-09-16
3. Simulated physiological stretch-induced proliferation of human bladder smooth muscle cells is regulated by MMPs;Archives of Biochemistry and Biophysics;2014-12
4. Tissue Engineering of Diseased Bladder using a Collagen Scaffold in a Bladder Exstrophy Model;BJU International;2013-12
5. This Month in Pediatric Urology;Journal of Urology;2012-10
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