Common developmental genome deprogramming in schizophrenia — Role of Integrative Nuclear FGFR1 Signaling (INFS)-Reference-Cited by-同舟云学术

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Common developmental genome deprogramming in schizophrenia — Role of Integrative Nuclear FGFR1 Signaling (INFS)

Published:2017-07 Issue: Volume:185 Page:17-32
ISSN:0920-9964
Container-title:Schizophrenia Research
language:en
Short-container-title:Schizophrenia Research

Author:

Narla S.T.,Lee Y-W.,Benson C.A.,Sarder P.,Brennand K.J.,Stachowiak E.K.,Stachowiak M.K.

Funder

New York State Department of Health

National Science Foundation

Patrick P. Lee Foundation

NIH

New York Stem Cell Foundation

Publisher

Elsevier BV

Subject

Biological Psychiatry,Psychiatry and Mental health

Reference100 articles.

1. Altered distribution of nicotinamide-adenine dinucleotide phosphate-diaphorase cells in frontal lobe of schizophrenics implies disturbances of cortical development;Akbarian;Arch. Gen. Psychiatry,1993

2. Further evidence of abnormal cytoarchitecture of the entorhinal cortex in schizophrenia using spatial point pattern analyses;Arnold;Biol. Psychiatry,1997

3. Cooperation of nuclear FGFR1 and Nurr1 offers a new interactive mechanism in postmitotic development of mesencephalic dopaminergic neurons;Baron;J. Biol. Chem.,2012

4. Cooperation of nuclear fibroblast growth factor receptor 1 and nurr1 offers new interactive mechanism in postmitotic development of mesencephalic dopaminergic neurons;Baron;J. Biol. Chem.,2012

5. MicroRNAs: target recognition and regulatory functions;Bartel;Cell,2009

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