The ability of guinea pigs to synthesize carnitine at a normal rate from ϵ-N-trimethyllysine or γ-butyrobetaine in vivo is not compromised by experimental vitamin C deficiency-Reference-Cited by-同舟云学术

The ability of guinea pigs to synthesize carnitine at a normal rate from ϵ-N-trimethyllysine or γ-butyrobetaine in vivo is not compromised by experimental vitamin C deficiency

Published:1995-05 Issue:5 Volume:44 Page:624-629
ISSN:0026-0495
Container-title:Metabolism
language:en
Short-container-title:Metabolism

Author:

Rebouche Charles J.

Publisher

Elsevier BV

Subject

Endocrinology,Endocrinology, Diabetes and Metabolism

Reference17 articles.

1. Ascorbic acid and carnitine biosynthesis;Rebouche;Am J Clin Nutr,1991

2. Effect of ascorbic acid deficiency on the in vivo synthesis of carnitine;Nelson;Biochim Biophys Acta,1981

3. Effect of vitamin C deficiency on hydroxylation of trimethylaminobutyrate to carnitine in the guinea pig;Thoma;Biochim Biophys Acta,1984

4. Role of the liver in carnitine metabolism: The mechanism of development of carnitine-deficient status in guinea-pigs;Alkonyi;J Clin Chem Clin Biochim,1990

5. Carnitine biosynthesis from γ-butyrobetaine and from exogenous protein-bound 6-N-trimethyl-l-lysine by the perfused guinea pig liver;Dunn,1984

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