Elamipretide Promotes Mitophagosome Formation and Prevents Its Reduction Induced by Nutrient Excess in INS1 β-cells-Reference-Cited by-同舟云学术

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Elamipretide Promotes Mitophagosome Formation and Prevents Its Reduction Induced by Nutrient Excess in INS1 β-cells

Published:2018-12 Issue:24 Volume:430 Page:4823-4833
ISSN:0022-2836
Container-title:Journal of Molecular Biology
language:en
Short-container-title:Journal of Molecular Biology

Author:

Petcherski Anton,Trudeau Kyle M.,Wolf Dane M.,Segawa Mayuko,Lee Jennifer,Taddeo Evan P.,Deeney Jude T.,Liesa Marc

Funder

UCSD/UCLA Diabetes Research Center

Publisher

Elsevier BV

Subject

Molecular Biology,Structural Biology

Reference28 articles.

1. Novel therapies targeting inner mitochondrial membrane—from discovery to clinical development;Szeto;Pharm. Res.,2011

2. Reduction of early reperfusion injury with the mitochondria-targeting peptide bendavia;Brown;J. Cardiovasc. Pharmacol. Ther.,2014

3. The mitochondrial-targeted compound SS-31 re-energizes ischemic mitochondria by interacting with cardiolipin;Birk;J. Am. Soc. Nephrol.,2013

4. Mitochondrial H2O2 emission and cellular redox state link excess fat intake to insulin resistance in both rodents and humans;Anderson;J. Clin. Invest.,2009

5. Mitochondrial targeting with antioxidant peptide SS-31 prevents mitochondrial depolarization, reduces islet cell apoptosis, increases islet cell yield, and improves posttransplantation function;Thomas;J. Am. Soc. Nephrol.,2007

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