Inhibiting EGFR Signaling Holds Promise for Treating Palmoplantar Keratodermas
Author:
Funder
Medical School, University of Michigan
National Institutes of Health
Publisher
Elsevier BV
Subject
Cell Biology,Dermatology,Molecular Biology,Biochemistry
Reference17 articles.
1. EGFR signaling is overactive in pachyonychia congenita: effective treatment with oral erlotinib;Basset;J Invest Dermatol,2022
2. iRHOM2-dependent regulation of ADAM17 in cutaneous disease and epidermal barrier function;Brooke;Hum Mol Genet,2014
3. Gene expression profiling in pachyonychia congenita skin;Cao;J Dermatol Sci,2015
4. TRP channel regulates EGFR signaling in hair morphogenesis and skin barrier formation;Cheng;Cell,2010
5. Use of epidermal growth factor receptor inhibitor erlotinib to treat palmoplantar keratoderma in patients with Olmsted syndrome caused by TRPV3 mutations;Greco;JAMA Dermatol,2020
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1. Significance of stress keratin expression in normal and diseased epithelia;iScience;2024-02
2. Pachyonychia Congenita: A Research Agenda Leading to New Therapeutic Approaches;Journal of Investigative Dermatology;2023-12
3. Posttranslational modifications of keratins and their associated proteins as therapeutic targets in keratin diseases;Current Opinion in Cell Biology;2023-12
4. Defective cathepsin Z affects EGFR expression and causes autosomal dominant palmoplantar keratoderma;British Journal of Dermatology;2023-05-20
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