Regnase-1, an Immunomodulator, Limits the IL-36/IL-36R Autostimulatory Loop in Keratinocytes to Suppress Skin Inflammation
Author:
Publisher
Elsevier BV
Subject
Cell Biology,Dermatology,Molecular Biology,Biochemistry
Reference15 articles.
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3. The IκB kinase complex regulates the stability of cytokine-encoding mRNA induced by TLR–IL-1R by controlling degradation of regnase-1;Iwasaki;Nat Immunol,2011
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5. A novel CCCH-zinc finger protein family regulates proinflammatory activation of macrophages;Liang;J Biol Chem,2008
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1. Keratinocyte and myeloid MCPIP1 have distinct roles in maintaining skin homeostasis;Biochimica et Biophysica Acta (BBA) - General Subjects;2024-10
2. The role of interleukin‐36 in health and disease states;Journal of the European Academy of Dermatology and Venereology;2024-05-23
3. Emerging Role of the IL-36/IL-36R Axis in Multiple Inflammatory Skin Diseases;Journal of Investigative Dermatology;2024-02
4. Depletion of Mcpip1 in murine myeloid cells results in intestinal dysbiosis followed by allergic inflammation;Biochimica et Biophysica Acta (BBA) - Molecular Basis of Disease;2023-10
5. Regnase-1 plays an essential role in maintaining skin immune homeostasis via regulation of chemokine expression;Biomedicine & Pharmacotherapy;2023-06
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