Senescent Dermal Fibroblasts Decrease Stemness in Basal Keratinocytes in a Bioengineered Model of Human Full-Thickness Skin-Reference-Cited by-同舟云学术

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Senescent Dermal Fibroblasts Decrease Stemness in Basal Keratinocytes in a Bioengineered Model of Human Full-Thickness Skin

Published:2024-07 Issue: Volume: Page:
ISSN:0022-202X
Container-title:Journal of Investigative Dermatology
language:en
Short-container-title:Journal of Investigative Dermatology

Author:

Low Evon^ORCID,Smith Lucy A.^ORCID,Miwa Satomi^ORCID,Fielder Edward^ORCID,Przyborski Stefan^ORCID,von Zglinicki Thomas^ORCID

Funder

Newcastle University

Publisher

Elsevier BV

Reference12 articles.

1. Regulation of cellular senescence by extracellular matrix during chronic fibrotic diseases;Blokland;Clin Sci (Lond),2020

2. Engineering a multilayered skin equivalent: the importance of endogenous extracellular matrix maturation to provide robustness and reproducibility;Costello;Methods Mol Biol,2019

3. A three-dimensional skin equivalent reflecting some aspects of in vivo aged skin;Diekmann;Exp Dermatol,2016

4. Effects of serially passaged fibroblasts on dermal and epidermal morphogenesis in human skin equivalents;Janson;Biogerontology,2013

5. Cellular senescence: a translational perspective;Kirkland;EBioMedicine,2017

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