GPR56/ADGRG1 Activation Promotes Melanoma Cell Migration via NTF Dissociation and CTF-Mediated Gα12/13/RhoA Signaling-Reference-Cited by-同舟云学术

登录注册会员服务联系我们

GPR56/ADGRG1 Activation Promotes Melanoma Cell Migration via NTF Dissociation and CTF-Mediated Gα12/13/RhoA Signaling

Published:2017-03 Issue:3 Volume:137 Page:727-736
ISSN:0022-202X
Container-title:Journal of Investigative Dermatology
language:en
Short-container-title:Journal of Investigative Dermatology

Author:

Chiang Nien-Yi,Peng Yen-Ming,Juang Horng-Heng,Chen Tse-Ching,Pan Hsiao-Lin,Chang Gin-Wen,Lin Hsi-Hsien

Publisher

Elsevier BV

Subject

Cell Biology,Dermatology,Molecular Biology,Biochemistry

Reference59 articles.

1. The adhesion GPCR Gpr56 regulates oligodendrocyte development via interactions with Galpha12/13 and RhoA;Ackerman;Nat Commun,2015

2. A novel evolutionarily conserved domain of cell-adhesion GPCRs mediates autoproteolysis;Arac;EMBO J,2012

3. The adhesion GPCRs: a unique family of G protein-coupled receptors with important roles in both central and peripheral tissues;Bjarnadottir;Cell Mol Life Sci,2007

4. The adhesion G protein-coupled receptor GPR56/ADGRG1 is an inhibitory receptor on human NK cells;Chang;Cell Rep,2016

5. Disease-associated GPR56 mutations cause bilateral frontoparietal polymicrogyria via multiple mechanisms;Chiang;J Biol Chem,2011

Cited by 38 articles. 订阅此论文施引文献订阅此论文施引文献，注册后可以免费订阅5篇论文的施引文献，订阅后可以查看论文全部施引文献

1. GPR56: A potential therapeutic target for neurological and psychiatric disorders;Biochemical Pharmacology;2024-08

2. Generic residue numbering of the GAIN domain of adhesion GPCRs;2024-07-19

3. GPR56/ADGRG1 induces biased Rho-ROCK-MLC and JAK-STAT3 signaling to promote amoeboid-like morphology and IL-6 upregulation in melanoma cells;2024-06-05

4. GPR56, an Adhesion GPCR with Multiple Roles in Human Diseases, Current Status and Future Perspective;Current Drug Targets;2024-06

5. Cytokine profiling and transcriptomics in mononuclear cells define immune variants in Meniere Disease;Genes & Immunity;2024-02-23

同舟云学术

1.学者识别学者识别

2.学术分析学术分析

3.人才评估人才评估

"同舟云学术"是以全球学者为主线，采集、加工和组织学术论文而形成的新型学术文献查询和分析系统，可以对全球学者进行文献检索和人才价值评估。用户可以通过关注某些学科领域的顶尖人物而持续追踪该领域的学科进展和研究前沿。经过近期的数据扩容，当前同舟云学术共收录了国内外主流学术期刊6万余种，收集的期刊论文及会议论文总量共计约1.5亿篇，并以每天添加12000余篇中外论文的速度递增。我们也可以为用户提供个性化、定制化的学者数据。欢迎来电咨询！咨询电话：010-8811{复制后删除}0370

www.globalauthorid.com

TOP

Copyright © 2019-2024 北京同舟云网络信息技术有限公司
京公网安备11010802033243号京ICP备18003416号-3