Uncoupling of ER/Mitochondrial Oxidative Stress in mTORC1 Hyperactivation-Associated Skin Hypopigmentation-Reference-Cited by-同舟云学术

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Uncoupling of ER/Mitochondrial Oxidative Stress in mTORC1 Hyperactivation-Associated Skin Hypopigmentation

Published:2018-03 Issue:3 Volume:138 Page:669-678
ISSN:0022-202X
Container-title:Journal of Investigative Dermatology
language:en
Short-container-title:Journal of Investigative Dermatology

Author:

Yang Fei,Yang Lingli,Wataya-Kaneda Mari,Yoshimura Takuji,Tanemura Atsushi,Katayama Ichiro

Funder

Ministry of Education, Culture, Sports, Science, and Technology of Japan

Japan Agency for Medical Research and Development

Clinical Research Project for Rare and Intractable Diseases

Publisher

Elsevier BV

Subject

Cell Biology,Dermatology,Molecular Biology,Biochemistry

Reference43 articles.

1. Inhibition of the phosphatidylinositol 3-kinase/p70(S6)-kinase pathway induces B16 melanoma cell differentiation;Busca;J Biol Chem,1996

2. Tuberous sclerosis complex inactivation disrupts melanogenesis via mTORC1 activation;Cao;J Clin Invest,2017

3. TSC-mTOR maintains quiescence and function of hematopoietic stem cells by repressing mitochondrial biogenesis and reactive oxygen species;Chen;J Exp Med,2008

4. The tuberous sclerosis complex;Crino;New Engl J Med,2006

5. Tuberous sclerosis complex: everything old is new again;Ess;J Neurodev Disord,2009

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