Membranal Expression of Calreticulin Induced by Unfolded Protein Response in Melanocytes: A Mechanism Underlying Oxidative Stress-induced Autoimmunity in Vitiligo
Author:
Funder
National Natural Science Foundation of China
Publisher
Elsevier BV
Subject
Cell Biology,Dermatology,Molecular Biology,Biochemistry
Reference47 articles.
1. Impact of Calreticulin and Its Mutants on Endoplasmic Reticulum Function in Health and Disease;Arshad;Prog Mol Subcell Biol,2021
2. Particulate Matter Promotes Melanin Production through Endoplasmic Reticulum Stress‒Mediated IRE1α Signaling;Ahn;J Invest Dermatol,2022
3. Systemic lupus erythematosus is associated with increased auto-antibody titers against calreticulin and grp94, but calreticulin is not the Ro/SS-A antigen;Boehm;Eur J Clin Invest,1994
4. Autoimmune vitiligo does not require the ongoing priming of naive CD8 T cells for disease progression or associated protection against melanoma;Byrne;J Immunol,2014
5. Immune Checkpoint Blockade Enhances Shared Neoantigen-Induced T-cell Immunity Directed against Mutated Calreticulin in Myeloproliferative Neoplasms;Cimen-Bozkus;Cancer Discov,2019
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