Unilateral AMPA lesions of nucleus basalis magnocellularis induce a sensorimotor deficit which is differentially altered by arecoline and nicotine

Author:

Abdulla F.A.,Calaminici M.-R.,Stephenson J.D.,Sinden J.D.

Publisher

Elsevier BV

Subject

Behavioral Neuroscience

Reference30 articles.

1. Changes in sensitivity of rat frontal cortex neurones to acetylcholine (ACh) after α-amino-3-oh-4-isoxozole propionic acid (AMPA) lesions of nucleus basalis magnocellularis and after embryonic basal forebrain transplants;Abdulla;Restorative Neurol. Neurosci.,1992

2. Chronic nicotine treatment and unilateral lesion of nucleus basalis (nb) with α-amino-3-oh-4-isoxozole propionic acid (AMPA) alter rat frontal cortex pyramidal cell sensitivity to nicotine;Abdulla;Br. J. Pharmacol.,1993

3. Abdulla, F.A., Calaminici, M.-R., Raevsky, V.V., Sinden, J.D., Gray, J.A. and Stephenson, J.D., An iontopheretic study of the effects of AMPA lesions of the nucleus basalis magnocellularis on cholinergic and GABAergic influences on frontal cortex neurones of rats, Exp. Brain Res., in press.

4. Effects of ventrolateral thalamic nucleus cooling on initiation of forelimb ballistic flexion movements by conditioned cats;Benita;Exp. Brain Res.,1979

5. Cholinergic projections from the basal forebrain to frontal parietal, temporal, occipital and cingulate cortices: a combined fluorescent tracer and acetylcholinesterase analysis;Bigl;Brain Res. Bull.,1982

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