Hypercysteinemia promotes atherosclerosis by reducing protein S-nitrosylation-Reference-Cited by-同舟云学术

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Hypercysteinemia promotes atherosclerosis by reducing protein S-nitrosylation

Published:2015-03 Issue: Volume:70 Page:253-259
ISSN:0753-3322
Container-title:Biomedicine & Pharmacotherapy
language:en
Short-container-title:Biomedicine & Pharmacotherapy

Author:

Chen Yulong,Liu Ruihan,Zhang Guangwei,Yu Qi,Jia Min,Zheng Chao,Wang Yanli,Xu Cangbao,Zhang Yaping,Liu Enqi

Funder

National Natural Science Foundation of China

Publisher

Elsevier BV

Subject

Pharmacology,General Medicine

Reference26 articles.

1. Nitrosylation: the prototypic redox-based signaling mechanism;Stamler;Cell,2001

2. Hypercontractile female hearts exhibit increased S-nitrosylation of the L-type Ca2+ channel alpha1 subunit and reduced ischemia/reperfusion injury;Sun;Circ Res,2006

3. Nitric oxide suppresses NADPH oxidase-dependent superoxide production by S-nitrosylation in human endothelial cells;Selemidis;Cardiovasc Res,2007

4. Nitric oxide promotes endothelial cell survival signaling through S-nitrosylation and activation of dynamin-2;Kang-Decker;J Cell Sci,2007

5. TNFalpha and oxLDL reduce protein S-nitrosylation in endothelial cells;Hoffmann;J Biol Chem,2001

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