TGF-β1 modulates podocyte migration by regulating the expression of integrin-β1 and -β3 through different signaling pathways-Reference-Cited by-同舟云学术

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TGF-β1 modulates podocyte migration by regulating the expression of integrin-β1 and -β3 through different signaling pathways

Published:2018-09 Issue: Volume:105 Page:974-980
ISSN:0753-3322
Container-title:Biomedicine & Pharmacotherapy
language:en
Short-container-title:Biomedicine & Pharmacotherapy

Author:

Chen Chien-An,Chang Jer-Ming,Chang Eddy-Essen,Chen Hung-Chun,Yang Yu-Lin

Funder

Tainan Sinlau Hospital

Nephrology Laboratory, Kaohsiung Medical University, Kaohsiung, Taiwan

Publisher

Elsevier BV

Subject

Pharmacology,General Medicine

Reference35 articles.

1. Podocytes undergo phenotypic changes and express macrophagic-associated markers in idiopathic collapsing glomerulopathy;Bariety;Kidney Int.,1998

2. The podocyte’s response to stress: the enigma of foot process effacement;Kriz;Am. J. Physiol. Ren. Physiol.,2013

3. Reduced podocyte expression of alpha3beta1 integrins and podocyte depletion in patients with primary focal segmental glomerulosclerosis and chronic PAN-treated rats;Chen;J. Lab. Clin. Med.,2006

4. Podocyte depletion causes glomerulosclerosis: diphtheria toxin-induced podocyte depletion in rats expressing human diphtheria toxin receptor transgene;Wharram;J. Am. Soc. Nephrol.,2005

5. Podocyte migration during nephrotic syndrome requires a coordinated interplay between cathepsin L and alpha3 integrin;Reiser;J. Biol. Chem.,2004

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3. Role of biophysics and mechanobiology in podocyte physiology;Nature Reviews Nephrology;2024-03-05

4. The potential role of differentially expressed tRNA-derived fragments in high glucose-induced podocytes;Renal Failure;2024-02-18

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