EG-VEGF silencing inhibits cell proliferation and promotes cell apoptosis in pancreatic carcinoma via PI3K/AKT/mTOR signaling pathway-Reference-Cited by-同舟云学术

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EG-VEGF silencing inhibits cell proliferation and promotes cell apoptosis in pancreatic carcinoma via PI3K/AKT/mTOR signaling pathway

Published:2019-01 Issue: Volume:109 Page:762-769
ISSN:0753-3322
Container-title:Biomedicine & Pharmacotherapy
language:en
Short-container-title:Biomedicine & Pharmacotherapy

Author:

Yan Xiaogang,Hui Yongfeng,Hua Yongqiang,Huang Liya,Wang Libin,Peng Fei,Tang Chaofeng,Liu Di,Song Jianjun,Wang Feng

Funder

School-level Project of Ningxia Medical University

Ningxia Natural Science Foundation

Scientific Research Project of Ningxia Medical University

Publisher

Elsevier BV

Subject

Pharmacology,General Medicine

Reference36 articles.

1. Pancreatic cancer;Vincent;Lancet,2011

2. Brain metastasis in pancreatic cancer;Lemke;Int. J. Mol. Sci.,2013

3. COX-2 over-expression correlates with VEGF and tumour angiogenesis in canine mammary cancer;Queiroga;Vet. J.,2011

4. Endocrine gland-derived vascular endothelial growth factor (EG-VEGF) and its receptor PROKR2 are associated to human colorectal cancer progression and peritoneal carcinomatosis;Benlahfid;Cancer Biomark.,2018

5. Upregulation of endocrine gland-derived vascular endothelial growth factor in papillary thyroid cancers displaying infiltrative patterns, lymph node metastases, and BRAF mutation;Pasquali;Thyroid,2011

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