Naringenin promoted spinal microglia M2 polarization in rat model of cancer-induced bone pain via regulating AMPK/PGC-1α signaling axis-Reference-Cited by-同舟云学术

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Naringenin promoted spinal microglia M2 polarization in rat model of cancer-induced bone pain via regulating AMPK/PGC-1α signaling axis

Published:2022-05 Issue: Volume:149 Page:112912
ISSN:0753-3322
Container-title:Biomedicine & Pharmacotherapy
language:en
Short-container-title:Biomedicine & Pharmacotherapy

Author:

Ge Meng-Meng,Li Dan-Yang,Wang Lu,Zhang Long-Qing,Liu Dai-Qiang,Tian Yu-Ke,Ye Da-Wei,Liu Zhi-Heng,Zhou Ya-Qun,Yang Hui

Funder

Sanming Project of Medicine in Shenzhen

National Natural Science Foundation of China

Publisher

Elsevier BV

Subject

Pharmacology,General Medicine

Reference59 articles.

1. Bone cancer pain: from mechanism to therapy;Mantyh;Curr. Opin. Support Palliat. Care,2014

2. Gabapentin normalizes spinal neuronal responses that correlate with behavior in a rat model of cancer-induced bone pain;Donovan-Rodriguez;Anesthesiology,2005

3. The role of spinal GABAB receptors in cancer-induced bone pain in rats;Zhou;J. Pain.,2017

4. The therapeutic potential of GABA in neuron-glia interactions of cancer-induced bone pain;Ge;Eur. J. Pharmacol.,2019

5. IL-18 contributes to bone cancer pain by regulating glia cells and neuron interaction;Liu;J. Pain.,2018

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4. Naringenin relieves paclitaxel‐induced pain by suppressing calcitonin gene‐related peptide signalling and enhances the anti‐tumour action of paclitaxel;British Journal of Pharmacology;2024-05-07

5. UPLC-Q-TOF-MS^E Based Profiling of the Chemical Composition of Different Parts of Tetrastigma hemsleyanum;Natural Product Communications;2024-05

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