Celastrol inhibits LL37-induced rosacea by inhibiting Ca2+/CaMKII-mTOR-NF-κB activation-Reference-Cited by-同舟云学术

Celastrol inhibits LL37-induced rosacea by inhibiting Ca2+/CaMKII-mTOR-NF-κB activation

Published:2022-09 Issue: Volume:153 Page:113292
ISSN:0753-3322
Container-title:Biomedicine & Pharmacotherapy
language:en
Short-container-title:Biomedicine & Pharmacotherapy

Author:

Zeng Qingyu,Yang Jin,Yan Guorong,Zhang Linglin,Wang Peiru,Zhang Haiyan,Chen Qi,Cao Yajing,Liu Xiaojing,Wang Xiuli

Publisher

Elsevier BV

Subject

Pharmacology,General Medicine

Reference35 articles.

1. Global epidemiology and clinical spectrum of rosacea, highlighting skin of color: Review and clinical practice experience;Alexis;J. Am. Acad. Dermatol.,2019

2. N2-Polarized Neutrophils Reduce Inflammation in Rosacea by Regulating Vascular Factors and Proliferation of CD4(+) T Cells;Zhao;J. Invest Dermatol,2021

3. A positive feedback loop between mTORC1 and cathelicidin promotes skin inflammation in rosacea;Deng;EMBO Mol. Med.,2021

4. Antimicrobial peptide LL-37 drives rosacea-like skin inflammation in an NLRP3-dependent manner;Yoon;J. Invest Dermatol.,2021

5. LL-37, the only human member of the cathelicidin family of antimicrobial peptides;Durr;Biochim Biophys. Acta,2006

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