Arginine homozygosity in codon 72 of p53 correlates with failure to imatinib response in chronic myeloid leukemia-Reference-Cited by-同舟云学术

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Arginine homozygosity in codon 72 of p53 correlates with failure to imatinib response in chronic myeloid leukemia

Published:2013-03 Issue:2 Volume:67 Page:103-107
ISSN:0753-3322
Container-title:Biomedicine & Pharmacotherapy
language:en
Short-container-title:Biomedicine & Pharmacotherapy

Author:

Camelo-Santos Jeany,do Prado Barbosa Adriana,de Paula Silveira-Lacerda Elisangela,Guillo Lidia Andreu

Publisher

Elsevier BV

Subject

Pharmacology,General Medicine

Reference38 articles.

1. Imatinib: a review of its use in chronic myeloid leukaemia;Moen;Drugs,2007

2. Mechanisms and novel approaches in overriding tyrosine kinase inhibitor resistance in chronic myeloid leukemia;Karvela;Expert Rev Anticancer Ther,2012

3. Regulation of the c-Abl and Bcr-Abl tyrosine kinases;Hanctschel;Nat Rev Mol Cell Biol,2004

4. Molecular biology of bcr-abl1-positive chronic myeloid leukemia;Quintás-Cardama;Blood,2009

5. The expanding options for front-line treatment of patients with newly diagnosed CML;Pinilla-Ibarz;Crit Rev Oncol Hematol,2012

Cited by 3 articles. 订阅此论文施引文献订阅此论文施引文献，注册后可以免费订阅5篇论文的施引文献，订阅后可以查看论文全部施引文献

1. Genetic association of TP53 Pro72Arg polymorphism (rs1042522) in Leukemia: An updated meta-analysis of 10 case-control studies;Human Gene;2022-12

2. Genetic Biomarkers in Chronic Myeloid Leukemia: What Have We Learned So Far?;International Journal of Molecular Sciences;2021-11-19

3. TP53 codon 72 polymorphism predicts chronic myeloid leukemia susceptibility and treatment outcome;Blood Cells, Molecules, and Diseases;2016-07

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