Inhibition of CACNA1H attenuates doxorubicin-induced acute cardiotoxicity by affecting endoplasmic reticulum stress-Reference-Cited by-同舟云学术

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Inhibition of CACNA1H attenuates doxorubicin-induced acute cardiotoxicity by affecting endoplasmic reticulum stress

Published:2019-12 Issue: Volume:120 Page:109475
ISSN:0753-3322
Container-title:Biomedicine & Pharmacotherapy
language:en
Short-container-title:Biomedicine & Pharmacotherapy

Author:

Hu Junxia,Wu Qi,Wang Zhiwei,Hong Junmou,Chen Ruoshi,Li Bowen,Hu Zhipeng,Hu Xiaoping,Zhang Min

Funder

National Natural Science Foundation of China

Key Support Project of Health Commission of Hubei Province

Guiding Fund of Renmin Hospital of Wuhan University

Publisher

Elsevier BV

Subject

Pharmacology,General Medicine

Reference33 articles.

1. Pharmacogenetics of target genes across doxorubicin disposition pathway: a review;Lal;Curr. Drug Metab.,2010

2. Amlodipine inhibits doxorubicin-induced apoptosis in neonatal rat cardiac myocytes;Yamanaka;J. Am. Coll. Cardiol.,2003

3. Anthracycline-related cardiotoxicity in childhood cancer survivors;Lipshultz;Curr. Opin. Cardiol.,2014

4. Doxorubicin cardiomyopathy;Chatterjee;Cardiology,2010

5. Isodunnianol alleviates doxorubicin-induced myocardial injury by activating protective autophagy;Chen;Food Funct.,2019

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4. The mmu_circ_003062, hsa_circ_0075663/miR-490-3p/CACNA1H axis mediates apoptosis in renal tubular cells in association with endoplasmic reticulum stress following ischemic acute kidney injury;International Immunopharmacology;2024-05

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