Exploration of the optimal dose of HOE-642 for the protection of neuronal mitochondrial function after cardiac arrest in rats-Reference-Cited by-同舟云学术

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Exploration of the optimal dose of HOE-642 for the protection of neuronal mitochondrial function after cardiac arrest in rats

Published:2019-02 Issue: Volume:110 Page:818-824
ISSN:0753-3322
Container-title:Biomedicine & Pharmacotherapy
language:en
Short-container-title:Biomedicine & Pharmacotherapy

Author:

Wei Lanying,Zhao Wenshuai,Hu Yanan,Wang Xifan,Liu Xintong,Zhang Pengjiao,Han Fei

Funder

Nature Science Foundation

Department of Science and Technology, Heilongjiang, China

Publisher

Elsevier BV

Subject

Pharmacology,General Medicine

Reference34 articles.

1. Mitochondrial dysfunction and oxidative stress as determinants of cell death/survival in stroke;Chan;Ann. N. Y. Acad. Sci.,2005

2. Nrf2 signaling in coordinated activation of antioxidant gene expression;Jaiswal;Free Radic. Biol. Med.,2004

3. Cariporide (HOE642), a selective Na+-H+ exchange inhibitor, inhibits the mitochondrial death pathway;Teshima;Circulation,2003

4. Inhibiting mitochondrial permeability transition pore opening: a new paradigm for myocardial preconditioning?;Hausenloy;Cardiovasc. Res.,2002

5. Gene inactivation of Na+/H+ exchanger isoform 1 attenuates apoptosis and mitochondrial damage following transient focal cerebral ischemia;Wang;Eur. J. Neurosci.,2008

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1. Vitamin E alleviates pyraclostrobin-induced toxicity in zebrafish (Danio rerio) and its potential mechanisms;Science of The Total Environment;2024-04

2. Prophylactic supplement with melatonin prevented the brain injury after cardiac arrest in rats;Scientific Reports;2023-11-16

3. Prophylactic supplement with melatonin prevented the brain injury after cardiac arrest in rats through SIRT3/CypD activation and PINK1/Parkin induced mitophagy;2023-07-27

4. The Remaining Conundrum of the Role of the Na+/H+ Exchanger Isoform 1 (NHE1) in Cardiac Physiology and Pathology: Can It Be Rectified?;Reviews in Cardiovascular Medicine;2022-08-15

5. Prostaglandin E1 attenuates post‑cardiac arrest myocardial dysfunction through inhibition of mitochondria‑mediated cardiomyocyte apoptosis;Molecular Medicine Reports;2020-12-02

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