Cyclic di-GMP incorporates the transcriptional factor FleQ03 in Pseudomonas syringae MB03 to elicit biofilm-dependent resistance in response to Caenorhabditis elegans predation
Author:
Funder
National Natural Science Foundation of China
Publisher
Elsevier BV
Reference48 articles.
1. The pathogenicity of Pseudomonas syringae MB03 against Caenorhabditis elegans and the transcriptional response of nematicidal genes upon different nutritional conditions;Ali;Front. Microbiol.,2016
2. Identification of the genes of the plant pathogen Pseudomonas syringae MB03 required for the nematicidal activity against Caenorhabditis elegans through an integrated approach;Ali;Front. Microbiol.,2022
3. The c-di-GMP phosphodiesterase BifA is involved in the virulence of bacteria from the Pseudomonas syringae complex;Aragón;Mol. Plant Pathol.,2015
4. The FleQ protein from Pseudomonas aeruginosa functions as both a repressor and an activator to control gene expression from the pel operon promoter in response to c-di-GMP;Baraquet;Nucleic Acids Res.,2012
5. Nematicidal effects of 2-methyl-aconitate isomerase from the phytopathogen Pseudomonas syringae MB03 on the model nematode Caenorhabditis elegans;Bashir;J. Invertebr. Pathol.,2021
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