Inactivated AMPK-α2 promotes the progression of diabetic brain damage by Cdk5 phosphorylation at Thr485 site-Reference-Cited by-同舟云学术

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Inactivated AMPK-α2 promotes the progression of diabetic brain damage by Cdk5 phosphorylation at Thr485 site

Published:2020-01 Issue: Volume:168 Page:277-284
ISSN:0300-9084
Container-title:Biochimie
language:en
Short-container-title:Biochimie

Author:

Li Yan,Xiang Qiong,Yao Yu-Han,Li Jing-Jing,Wang Yan,Li Xian-Hui

Funder

National Science Foundation of China

Education Department of Hunan Province

OCPC-Postdoctoral fund program

Jishou University

Publisher

Elsevier BV

Subject

General Medicine,Biochemistry

Reference32 articles.

1. Regulated proteolysis of RAGE and AbetaPP as possible link between type 2 diabetes mellitus and Alzheimer’s disease;Kojro;J. Alzheimer’s Dis.,2009

2. Roles of AMP-activated protein kinase in Alzheimer’s disease;Cai;NeuroMolecular Med.,2012

3. An integrated and unifying hypothesis for the metabolic basis of sporadic Alzheimer’s disease;Erol;J. Alzheimer’s Dis.,2008

4. AMPK: a metabolic gauge regulating whole-body energy homeostasis;Lage;Trends Mol. Med.,2008

5. AMPK in health and disease;Steinberg;Physiol. Rev.,2009

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