Sodium, calcium and exocytosis: Confessions of calcified scientists
Author:
Publisher
Elsevier BV
Subject
Physiology (medical),General Neuroscience
Reference33 articles.
1. On the role of mitochondria in transmitter release from motor nerve terminals;Alnaes;J Physiol (Lond),1975
2. Long-term synaptic facilitation during sodium accumulation in nerve terminals;Atwood;Brain Res,1975
3. Calcium action in synaptic transmitter release;Augustine;Annu Rev Neurosci,1987
4. A note on the mechanisms by which inhibitors of the sodium pump accelerate spontaneous release of transmitter from motor nerve terminals;Baker;J Physiol (Lond),1975
5. The action of sodium pump inhibitors on neuromuscular transmission;Birks,1968
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1. Ouabain evokes exocytosis dependent on ryanodine and mitochondrial calcium stores that is not followed by compensatory endocytosis at the neuromuscular junction;Neurochemistry International;2009-11
2. Pepsinogen secretion: coupling of exocytosis visualized by video microscopy and [Ca2+]iin single cells;American Journal of Physiology-Gastrointestinal and Liver Physiology;1998-06-01
3. Sodium-dependent increase in quantal secretion induced by brevetoxin-3 in Ca 2+ -free medium is associated with depletion of synaptic vesicles and swelling of motor nerve terminals in situ;Neuroscience;1997-03
4. Involvement of extracellular sodium in agonist-induced gonadotropin release from goldfish (Carassius auratus) gonadotrophs;ENDOCRINOLOGY;1996
5. Purine and pyrimidine nucleotides activate distinct signalling pathways in PC12 cells;Journal of Neuroscience Research;1995-08-15
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