RecA433 cells are defective in recF-mediated processing of disrupted replication forks but retain recBCD-mediated functions
Author:
Publisher
Elsevier BV
Subject
Health, Toxicology and Mutagenesis,Genetics,Molecular Biology
Reference53 articles.
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2. Mutants of Escherichia coli K-12 defective in DNA repair and in genetic recombination;Howard-Flanders;Genetics,1966
3. Abnormal metabolic response to ultraviolet light of a recombination deficient mutant of Escherichia coli K12;Clark;J. Mol. Biol.,1966
4. 3′ homologous free ends are required for stable joint molecule formation by the RecA and single-stranded binding proteins of Escherichia coli;Konforti;Proc. Natl. Acad. Sci. U.S.A.,1987
5. Homologous pairing in genetic recombination: recA protein makes joint molecules of gapped circular DNA and closed circular DNA;Cunningham;Cell,1980
Cited by 3 articles. 订阅此论文施引文献 订阅此论文施引文献，注册后可以免费订阅5篇论文的施引文献，订阅后可以查看论文全部施引文献
1. Replication Stress-Induced Genome Instability: The Dark Side of Replication Maintenance by Homologous Recombination;Journal of Molecular Biology;2013-11
2. Homologous Recombination as a Replication Fork Escort: Fork-Protection and Recovery;Biomolecules;2012-12-27
3. Involvement of recF in 254 nm Ultraviolet Radiation Resistance in Deinococcus radiodurans and Escherichia coli;Current Microbiology;2010-04-13
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