The RAD51 S181P mutation shortens lifespan of female mice-Reference-Cited by-同舟云学术

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The RAD51 S181P mutation shortens lifespan of female mice

Published:2024-07 Issue: Volume:829 Page:111878
ISSN:0027-5107
Container-title:Mutation Research - Fundamental and Molecular Mechanisms of Mutagenesis
language:en
Short-container-title:Mutation Research - Fundamental and Molecular Mechanisms of Mutagenesis

Author:

Dodds Sherry G.,Hubbard Gene,Choi Yong Jun,Myung Kyungjae,Elliot Gene,Garrett Lisa,Kim Tae Moon,Hasty Paul

Funder

Korea Industrial Technology Association

National Institutes of Health

National Human Genome Research Institute

Edward P. Evans Foundation

Institute for Basic Science

Cancer Prevention and Research Institute of Texas

Publisher

Elsevier BV

Reference26 articles.

1. Double-strand break repair-independent role for BRCA2 in blocking stalled replication fork degradation by MRE11;Schlacher;Cell,2011

2. A distinct replication fork protection pathway connects fanconi anemia tumor suppressors to RAD51-BRCA1/2;Schlacher;Cancer Cell,2012

3. Nearby inverted repeats fuse to generate acentric and dicentric palindromic chromosomes by a replication template exchange mechanism;Mizuno;Genes Dev.,2009

4. Recombination-restarted replication makes inverted chromosome fusions at inverted repeats;Mizuno;Nature,2012

5. Hydroxyurea-stalled replication forks become progressively inactivated and require two different RAD51-mediated pathways for restart and repair;Petermann;Mol. Cell,2010

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