Proanthocyanidin exposure to B6C3F1 mice significantly attenuates dimethylnitrosamine-induced liver tumor induction and mortality by differentially modulating programmed and unprogrammed cell deaths-Reference-Cited by-同舟云学术

Proanthocyanidin exposure to B6C3F1 mice significantly attenuates dimethylnitrosamine-induced liver tumor induction and mortality by differentially modulating programmed and unprogrammed cell deaths

Published:2005-11 Issue:1-2 Volume:579 Page:81-106
ISSN:0027-5107
Container-title:Mutation Research/Fundamental and Molecular Mechanisms of Mutagenesis
language:en
Short-container-title:Mutation Research/Fundamental and Molecular Mechanisms of Mutagenesis

Author:

Ray Sidhartha D.,Parikh Hemal,Bagchi Debasis

Publisher

Elsevier BV

Subject

Health, Toxicology and Mutagenesis,Genetics,Molecular Biology

Reference51 articles.

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4. Species specificity in nitrosamine carcinogenesis;Lijinsky;Basic Life Sci.,1983

5. Possible role of certain antioxidant enzymes in dimethylnitrosamine induced liver carcinogenesis;Liotti;Int. J. Cancer,1988

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