Chronic treatment with ATR and CHK1 inhibitors does not substantially increase the mutational burden of human cells-Reference-Cited by-同舟云学术

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Chronic treatment with ATR and CHK1 inhibitors does not substantially increase the mutational burden of human cells

Published:2023-07 Issue: Volume:827 Page:111834
ISSN:0027-5107
Container-title:Mutation Research/Fundamental and Molecular Mechanisms of Mutagenesis
language:en
Short-container-title:Mutation Research/Fundamental and Molecular Mechanisms of Mutagenesis

Author:

Casimir Lisa,Zimmer Samuel,Racine-Brassard Félix,Goudreau Félix,Jacques Pierre-Étienne,Maréchal Alexandre^ORCID

Funder

Fonds de Recherche du Québec - Santé

Alliance de recherche numérique du Canada

Canadian Institutes of Health Research

Natural Sciences and Engineering Research Council of Canada

Publisher

Elsevier BV

Subject

Health, Toxicology and Mutagenesis,Genetics,Molecular Biology

Reference126 articles.

1. ATR prohibits replication catastrophe by preventing global exhaustion of RPA;Toledo;Cell,2013

2. DNA – protein crosslink repair: proteases as DNA repair enzymes;Stingele;Trends Biochem. Sci.,2014

3. R loops and their composite cancer connections;Wells;Trends Cancer,2019

4. Causes and consequences of replication stress;Zeman;Nat. Cell Biol.,2013

5. Functional uncoupling of MCM helicase and DNA polymerase activities activates the ATR-dependent checkpoint;Byun;Genes Dev.,2005

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