Gintonin mitigates experimental autoimmune encephalomyelitis by stabilization of Nrf2 signaling via stimulation of lysophosphatidic acid receptors-Reference-Cited by-同舟云学术

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Gintonin mitigates experimental autoimmune encephalomyelitis by stabilization of Nrf2 signaling via stimulation of lysophosphatidic acid receptors

Published:2021-03 Issue: Volume:93 Page:384-398
ISSN:0889-1591
Container-title:Brain, Behavior, and Immunity
language:en
Short-container-title:Brain, Behavior, and Immunity

Author:

Choi Jong Hee,Oh Jinhee,Lee Min Jung,Ko Seong-Gyu,Nah Seung-Yeol,Cho Ik-Hyun

Funder

National Research Foundation of Korea

Publisher

Elsevier BV

Subject

Behavioral Neuroscience,Endocrine and Autonomic Systems,Immunology

Reference68 articles.

1. New insights into the role of oxidative stress mechanisms in the pathophysiology and treatment of multiple sclerosis;Adamczyk;Oxid. Med. Cell Longev.,2016

2. The role of immune regulatory molecules in multiple sclerosis;Afshar;J. Neuroimmunol.,2019

3. Disruption of central nervous system barriers in multiple sclerosis;Alvarez;BBA,2011

4. Multiple sclerosis: genetics, biomarkers, treatments;Axisa;Curr. Opin. Neurol.,2016

5. Lysophosphatidic acid receptor activation affects the C13NJ microglia cell line proteome leading to alterations in glycolysis, motility, and cytoskeletal architecture;Bernhart;Proteomics,2010

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