Tumor necrosis factor-mediated downregulation of spinal astrocytic connexin43 leads to increased glutamatergic neurotransmission and neuropathic pain in mice-Reference-Cited by-同舟云学术

Tumor necrosis factor-mediated downregulation of spinal astrocytic connexin43 leads to increased glutamatergic neurotransmission and neuropathic pain in mice

Published:2015-10 Issue: Volume:49 Page:293-310
ISSN:0889-1591
Container-title:Brain, Behavior, and Immunity
language:en
Short-container-title:Brain, Behavior, and Immunity

Author:

Morioka Norimitsu,Zhang Fang Fang,Nakamura Yoki,Kitamura Tomoya,Hisaoka-Nakashima Kazue,Nakata Yoshihiro

Funder

Grant-in-Aid for Scientific Research

Publisher

Elsevier BV

Subject

Behavioral Neuroscience,Endocrine and Autonomic Systems,Immunology

Reference76 articles.

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4. Connexin43, the major gap junction protein of astrocytes, is down-regulated in inflamed white matter in an animal model of multiple sclerosis;Brand-Schieber;J. Neurosci. Res.,2005

5. Connexin-43 induces chemokine release from spinal cord astrocytes to maintain late-phase neuropathic pain in mice;Chen;Brain,2014

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