Spinal interleukin-6 contributes to central sensitisation and persistent pain hypersensitivity in a model of juvenile idiopathic arthritis-Reference-Cited by-同舟云学术

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Spinal interleukin-6 contributes to central sensitisation and persistent pain hypersensitivity in a model of juvenile idiopathic arthritis

Published:2020-11 Issue: Volume:90 Page:145-154
ISSN:0889-1591
Container-title:Brain, Behavior, and Immunity
language:en
Short-container-title:Brain, Behavior, and Immunity

Author:

Kwok Charlie H.T.,Learoyd Annastazia E,Canet-Pons Julia,Trang Tuan,Fitzgerald Maria

Funder

Canadian Institutes of Health Research

Versus Arthritis

Publisher

Elsevier BV

Subject

Behavioral Neuroscience,Endocrine and Autonomic Systems,Immunology

Reference51 articles.

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2. Effective gene expression in the rat dorsal root ganglia with a non-viral vector delivered via spinal nerve injection;Chang;Sci. Rep.,2016

3. Quantitative assessment of tactile allodynia in the rat paw;Chaplan;J. Neurosci. Methods,1994

4. Cytokine Pathways and Joint Inflammation in Rheumatoid Arthritis;Choy;N. Engl. J. Med.,2001

5. Paediatric rheumatology: Juvenile idiopathic arthritis—are biologic agents effective for pain?;Consolaro;Nat. Rev. Rheumatol.,2013

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4. Valproate attenuates somatic hyperalgesia induced by orofacial inflammation combined with stress through inhibiting spinal IL-6 and STAT1 phosphorylation;Brain Research Bulletin;2024-03

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