Upregulation of CCL2 via ATF3/c-Jun interaction mediated the Bortezomib-induced peripheral neuropathy-Reference-Cited by-同舟云学术

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Upregulation of CCL2 via ATF3/c-Jun interaction mediated the Bortezomib-induced peripheral neuropathy

Published:2016-03 Issue: Volume:53 Page:96-104
ISSN:0889-1591
Container-title:Brain, Behavior, and Immunity
language:en
Short-container-title:Brain, Behavior, and Immunity

Author:

Liu Cuicui,Luan Shuo,OuYang Handong,Huang Zhenzhen,Wu Shaoling,Ma Chao,Wei Jiayou,Xin Wenjun

Funder

National Natural Science Foundation of China

Publisher

Elsevier BV

Subject

Behavioral Neuroscience,Endocrine and Autonomic Systems,Immunology

Reference45 articles.

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2. The proteasome: a suitable antineoplastic target;Adams;Nat. Rev. Cancer,2004

3. Proteasome inhibitors: a novel class of potent and effective antitumor agents;Adams;Cancer Res.,1999

4. Neuronal CC chemokines: the distinct roles of CCL21 and CCL2 in neuropathic pain;Biber;Front. Cell. Neurosci.,2014

5. Genome-wide siRNA screen for modulators of cell death induced by proteasome inhibitor bortezomib;Chen;Cancer Res.,2010

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