TRIF is a key inflammatory mediator of acute sickness behavior and cancer cachexia-Reference-Cited by-同舟云学术

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TRIF is a key inflammatory mediator of acute sickness behavior and cancer cachexia

Published:2018-10 Issue: Volume:73 Page:364-374
ISSN:0889-1591
Container-title:Brain, Behavior, and Immunity
language:en
Short-container-title:Brain, Behavior, and Immunity

Author:

Burfeind Kevin G.,Zhu Xinxia,Levasseur Peter R.,Michaelis Katherine A.,Norgard Mason A.,Marks Daniel L.

Funder

National Institutes of Health

Brenden-Colson Center for Pancreatic Care

Publisher

Elsevier BV

Subject

Behavioral Neuroscience,Endocrine and Autonomic Systems,Immunology

Reference66 articles.

1. Wasting as independent risk factor for mortality in chronic heart failure;Anker;Lancet,1997

2. Consensus on cachexia definitions;Argiles;J. Am. Med. Dir. Assoc.,2010

3. Intravascular inflammation triggers intracerebral activated microglia and contributes to secondary brain injury after experimental subarachnoid hemorrhage (eSAH);Atangana;Transl. Stroke Res.,2017

4. Cachexia worsens prognosis in patients with resectable pancreatic cancer;Bachmann;J. Gastrointest. Surg.,2008

5. Assessing mouse behaviour throughout the light/dark cycle using automated in-cage analysis tools;Bains;J. Neurosci. Methods,2017

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