The MHC class II transactivator modulates seeded alpha-synuclein pathology and dopaminergic neurodegeneration in an in vivo rat model of Parkinson's disease

Author:

Jimenez-Ferrer Itzia,Bäckström Filip,Dueñas-Rey Alfredo,Jewett Michael,Boza-Serrano Antonio,Luk Kelvin C.,Deierborg Tomas,Swanberg Maria

Funder

Kungliga Fysiografiska Sällskapet i Lund

Magnus Bergvalls Stiftelse

Åke Wiberg Stiftelse

Vetenskapsrådet

Publisher

Elsevier BV

Subject

Behavioral Neuroscience,Endocrine and Autonomic Systems,Immunology

Reference46 articles.

1. Staging of brain pathology related to sporadic Parkinson's disease;Braak;Neurobiol. Aging,2003

2. Infiltration of CD4+ lymphocytes into the brain contributes to neurodegeneration in a mouse model of Parkinson disease;Brochard;J. Clin. Invest.,2009

3. Prying into the Prion Hypothesis for Parkinson's disease;Brundin;J. Neurosci.,2017

4. Mice lacking the MHC class II transactivator (CIITA) show tissue-specific impairment of MHC class II expression;Chang;Immunity,1996

5. Microglial inflammation in the parkinsonian substantia nigra: relationship to alpha-synuclein deposition;Croisier;J. Neuroinflam.,2005

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