TNF-α from hippocampal microglia induces working memory deficits by acute stress in mice-Reference-Cited by-同舟云学术

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TNF-α from hippocampal microglia induces working memory deficits by acute stress in mice

Published:2016-07 Issue: Volume:55 Page:17-24
ISSN:0889-1591
Container-title:Brain, Behavior, and Immunity
language:en
Short-container-title:Brain, Behavior, and Immunity

Author:

Ohgidani Masahiro,Kato Takahiro A.,Sagata Noriaki,Hayakawa Kohei,Shimokawa Norihiro,Sato-Kasai Mina,Kanba Shigenobu

Funder

Japan Society for the Promotion of Science

Ministry of Education, Culture, Sports, Science, and Technology

AMED

Japanese Ministry of Health, Labour and Welfare

Kyushu University

Takeda Medical Research Foundation

SENSHIN Medical Research Foundation

Publisher

Elsevier BV

Subject

Behavioral Neuroscience,Endocrine and Autonomic Systems,Immunology

Reference37 articles.

1. Inflammasome-induced IL-1 beta secretion in microglia is characterized by delayed kinetics and is only partially dependent on inflammatory caspases;Burm;J. Neurosci.,2015

2. The concepts of stress and stress system disorders – Overview of physical and behavioral homeostasis;Chrousos;J. Am. Med. Assoc.,1992

3. Acute stress impairs spatial memory in male but not female rats: influence of estrous cycle;Conrad;Pharmacol. Biochem. Behav.,2004

4. Peripheral administration of an anti-TNF-alpha receptor fusion protein counteracts the amyloid induced elevation of hippocampal TNF-alpha levels and memory deficits in mice;Detrait;Neurochem. Int.,2014

5. TNF defined as a therapeutic target for rheumatoid arthritis and other autoimmune diseases (vol 9, pg 1245, 2003);Feldmann;Nat. Med.,2003

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