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Neuronal NR2B-containing NMDA receptor mediates spinal astrocytic c-Jun N-terminal kinase activation in a rat model of neuropathic pain

  • Published:2011-10 Issue:7 Volume:25 Page:1355-1366
  • ISSN:0889-1591
  • Container-title:Brain, Behavior, and Immunity
  • language:en
  • Short-container-title:Brain, Behavior, and Immunity

Author:

Wang Wei,Mei Xiao-Peng,Wei Yan-Yan,Zhang Ming-Ming,Zhang Ting,Wang Wen,Xu Li-Xian,Wu Sheng-Xi,Li Yun-Qing

Funder

National Natural Science Foundation of China

National Program of Basic Research of China

Program for Innovation Research Team Program of Ministry of Education of China

Publisher

Elsevier BV

Subject

Behavioral Neuroscience,Endocrine and Autonomic Systems,Immunology

Reference40 articles.

1. Fyn kinase-mediated phosphorylation of NMDA receptor NR2B subunit at Tyr1472 is essential for maintenance of neuropathic pain;Abe;Eur. J. NeuroSci.,2005

2. Cellular and molecular mechanisms of pain;Basbaum;Cell,2009

3. Role of the c-Jun N-terminal kinase pathway in retinal excitotoxicity, and neuroprotection by its inhibition;Bessero;J. Neurochem.,2010

4. A peptide inhibitor of c-Jun N-terminal kinase protects against excitotoxicity and cerebral ischemia;Borsello;Nat. Med.,2003

5. Induction of glial fibrillary acidic protein expression in astrocytes by nitric oxide;Brahmachari;J. Neurosci.,2006

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