Bidirectional brain-gut interactions and chronic pathological changes after traumatic brain injury in mice-Reference-Cited by-同舟云学术

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Bidirectional brain-gut interactions and chronic pathological changes after traumatic brain injury in mice

Published:2017-11 Issue: Volume:66 Page:56-69
ISSN:0889-1591
Container-title:Brain, Behavior, and Immunity
language:en
Short-container-title:Brain, Behavior, and Immunity

Author:

Ma Elise L.,Smith Allen D.,Desai Neemesh,Cheung Lumei,Hanscom Marie,Stoica Bogdan A.,Loane David J.,Shea-Donohue Terez,Faden Alan I.

Funder

National Institutes of Health

USDA CRIS

Publisher

Elsevier BV

Subject

Behavioral Neuroscience,Endocrine and Autonomic Systems,Immunology

Reference74 articles.

1. The systemic response to CNS injury;Anthony;Exp. Neurol.,2014

2. Host-dependent zonulin secretion causes the impairment of the small intestine barrier function after bacterial exposure;Asmar;Gastroenterology,2002

3. Repeated mild traumatic brain injury causes chronic neuroinflammation, changes in hippocampal synaptic plasticity, and associated cognitive deficits;Aungst;J. Cereb. Blood Flow Metab.,2014

4. Traumatic Brain Injury and Intestinal Dysfunction: Uncovering the Neuro-Enteric Axis;Bansal;J Neurotrauma,2009

5. Commensal microbiota affects ischemic stroke outcome by regulating intestinal γδ T cells;Benakis;Nat. Med.,2016

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