CRISPR/Cas9-mediated genome editing of the thymidine kinase gene in a clinical HSV-1 isolate identifies F289S as novel acyclovir-resistant mutation-Reference-Cited by-同舟云学术

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CRISPR/Cas9-mediated genome editing of the thymidine kinase gene in a clinical HSV-1 isolate identifies F289S as novel acyclovir-resistant mutation

Published:2024-08 Issue: Volume:228 Page:105950
ISSN:0166-3542
Container-title:Antiviral Research
language:en
Short-container-title:Antiviral Research

Author:

Zheng Shuxuan,Verjans Georges M.G.M.,Evers Anouk,van den Wittenboer Ellen,Tjhie Jeroen H.T.,Snoeck Robert,Wiertz Emmanuel J.H.J.,Andrei Graciela,van Kampen Jeroen J.A.,Lebbink Robert Jan^ORCID

Publisher

Elsevier BV

Reference27 articles.

1. Highly reliable heterologous system for evaluating resistance of clinical herpes simplex virus isolates to nucleoside analogues;Bestman-Smith;J. Virol.,2001

2. Relevance of non-synonymous thymidine kinase mutations for antiviral resistance of recombinant herpes simplex virus type 2 strains;Brunnemann;Antivir. Res.,2018

3. Genotypic characterization of UL23 thymidine kinase and UL30 DNA polymerase of clinical isolates of herpes simplex virus: natural polymorphism and mutations associated with resistance to antivirals;Burrel;Antimicrob. Agents Chemother.,2010

4. A new tool linking human cytomegalovirus drug resistance mutations to resistance phenotypes;Chevillotte;Antivir. Res.,2010

5. Molecular analysis of clinical isolates of acyclovir resistant herpes simplex virus;Chibo;Antivir. Res.,2004

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1. Aciclovir;Reactions Weekly;2024-08-10

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