Agonistic 4-1BB antibody fails to reduce disease burden during acute respiratory syncytial virus (RSV) infection
Author:
Funder
NIH
SickKids Foundation
Institute for Human Development, Child and Youth Health – CIHR
Publisher
Elsevier BV
Subject
Virology,Pharmacology
Reference23 articles.
1. A switch in costimulation from CD28 to 4-1BB during primary versus secondary CD8 T cell response to influenza in vivo;Bertram;J. Immunol.,2004
2. Protective T cell immunity against respiratory syncytial virus is efficiently induced by recombinant BCG;Bueno;Proc. Natl. Acad. Sci. U. S. A.,2008
3. Resistance to respiratory syncytial virus (RSV) challenge induced by infection with a vaccinia virus recombinant expressing the RSV M2 protein (Vac-M2) is mediated by CD8+ T cells, while that induced by Vac-F or Vac-G recombinants is mediated by antibodies;Connors;J. Virol.,1992
4. Respiratory syncytial viral infection in children with compromised immune function;Hall;N. Engl. J. Med.,1986
5. 4-1BB triggering ameliorates experimental autoimmune encephalomyelitis by modulating the balance between Th17 and regulatory T cells;Kim;J. Immunol.,2011
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