1. Prion Diseases,1997
2. Ecosystems supporting clusters of sporadic TSEs demonstrate excesses of the radical generating divalent cation, manganese, and deficiencies of antioxidant co factors Cu, Se, Fe, Zn. Does a foreign cation substitution at Prp’s Cu domain initiate TSE?;Purdey;Med. Hypotheses,2000
3. Does an ultra violet photoxidization of the manganese loaded/copper depleted prion protein in the retina initiate the pathogenesis of TSE?;Purdey;Med. Hypotheses,2001
4. High dose exposure to systemic phosmet insecticide modifies the phosphatidylinositol anchor on the prion protein; the origins of new variant transmissible spongiform encephalopathy?;Purdey;Med. Hypotheses,1998
5. The BSE Inquiry, Volumes 2/5/9/10, The Stationary Office Limited, PO Box 29, Norwich NR3 1GN, October 2000