Capsaicin inhibits the production of tumor necrosis factor α by LPS-stimulated murine macrophages, RAW 264.7: a PPARγ ligand-like action as a novel mechanism-Reference-Cited by-同舟云学术

Capsaicin inhibits the production of tumor necrosis factor α by LPS-stimulated murine macrophages, RAW 264.7: a PPARγ ligand-like action as a novel mechanism

Published:2004-07-14 Issue:1-3 Volume:572 Page:266-270
ISSN:0014-5793
Container-title:FEBS Letters
language:en
Short-container-title:

Author:

Park Jun-Young,Kawada Teruo,Han In-Seob,Kim Byung-Sam,Goto Tsuyoshi,Takahashi Nobuyuki,Fushiki Tohru,Kurata Tadao,Yu Rina

Publisher

Wiley

Subject

Cell Biology,Genetics,Molecular Biology,Biochemistry,Structural Biology,Biophysics

Reference25 articles.

1. Molecular mechanisms underlying chemopreventive activities of anti-inflammatory phytochemicals: down-regulation of COX-2 and iNOS through suppression of NF-κB activation

2. The capsaicin receptor: a heat-activated ion channel in the pain pathway

3. Capsaicin exhibits anti-inflammatory property by inhibiting IkB-a degradation in LPS-stimulated peritoneal macrophages

4. Expression of the peroxisome proliferator-activated receptor (PPAR ) in human atherosclerosis and regulation in macrophages by colony stimulating factors and oxidized low density lipoprotein

5. PPARγ: a Nuclear Regulator of Metabolism, Differentiation, and Cell Growth

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