Diabetes mellitus due to the toxic misfolding of proinsulin variants
Author:
Funder
American Diabetes Association and National Institutes of Health
Publisher
Wiley
Subject
Cell Biology,Genetics,Molecular Biology,Biochemistry,Structural Biology,Biophysics
Link
http://onlinelibrary.wiley.com/wol1/doi/10.1016/j.febslet.2013.04.044/fullpdf
Reference140 articles.
1. Proinsulin misfolding and diabetes: mutant INS gene-induced diabetes of youth;Liu;Trends Endocrinol. Metab.,2010
2. Neonatal diabetes mellitus: a model for personalized medicine;Greeley;Trends Endocrinol. Metab.,2010
3. Neonatal diabetes: an expanding list of genes allows for improved diagnosis and treatment;Greeley;Curr. Diab. Rep.,2011
4. Mutant proinsulin proteins associated with neonatal diabetes are retained in the endoplasmic reticulum and not efficiently secreted;Park;Biochem. Biophys. Res. Commun.,2010
5. Mutant INS-gene induced diabetes of youth: proinsulin cysteine residues impose dominant-negative inhibition on nonmutant proinsulin transport;Liu;PLoS ONE,2010
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