Overexpression of proadrenomedullin N-terminal 20 peptide blunts blood pressure rise and attenuates myocardial hypertrophy and fibrosis in hypertensive rats-Reference-Cited by-同舟云学术

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Overexpression of proadrenomedullin N-terminal 20 peptide blunts blood pressure rise and attenuates myocardial hypertrophy and fibrosis in hypertensive rats

Published:2005-08-15 Issue:22 Volume:579 Page:4997-5001
ISSN:0014-5793
Container-title:FEBS Letters
language:en
Short-container-title:

Author:

Cao Yuan-Ning,Kuwasako Kenji,Kato Johji,Nishihira Kensaku,Asada Yujiro,Eto Tanenao,Kitamura Kazuo

Publisher

Wiley

Subject

Cell Biology,Genetics,Molecular Biology,Biochemistry,Structural Biology,Biophysics

Reference22 articles.

1. Adrenomedullin: a novel hypotensive peptide isolated from human pheochromocytoma;Kitamura;Biochem. Biophys. Res. Commun.,1993

2. Direct cardiac and vascular actions of adrenomedullin in conscious sheep;Parkes;Br. J. Pharmacol.,1997

3. Autocrine/paracrine role of adrenomedullin in cultured endothelial and mesangial cells;Michibata;Kidney Int.,1998

4. Adrenomedullin and nitric oxide inhibit human endothelial cell apoptosis via a cyclic GMP-independent mechanism;Sata;Hypertension,2000

5. Inhibition of aldosterone production by adrenomedullin, a hypotensive peptide, in the rat;Yamaguchi;Hypertension,1996

Cited by 1 articles. 订阅此论文施引文献订阅此论文施引文献，注册后可以免费订阅5篇论文的施引文献，订阅后可以查看论文全部施引文献

1. Genetic loss of proadrenomedullin N-terminal 20 peptide (PAMP) in mice is compatible with survival;Peptides;2019-02

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